Salars in Qinghai live on both banks of the Yellow river, south and north, the northern ones are called Hualong or Bayan Salars while the southern ones are called Xunhua Salars. The region north of the Yellow river is a mix of discontinuous Salar and Tibetan villages while the region south of the yellow river is solidly Salar with no gaps in between, since Hui and Salars pushed the Tibetans on the south region out earlier. Tibetan women who converted to Islam were taken as wives on both banks of the river by Salar men. The term for maternal uncle (ajiu) is used for Tibetans by Salars since the Salars have maternal Tibetan ancestry. Tibetans witness Salar life passages in Kewa, a Salar village and Tibetan butter tea is consumed by Salars there as well. Other Tibetan cultural influences like Salar houses having four corners with a white stone on them became part of Salar culture as long as they were not prohibited by Islam. Hui people started assimilating and intermarrying with Salars in Xunhua after migrating there from Hezhou in Gansu due to the Chinese Ming dynasty ruling the Xunhua Salars after 1370 and Hezhou officials governed Xunhua. Many Salars with the Ma surname appear to be of Hui descent since a lot of Salars now have the Ma surname while in the beginning the majority of Salars had the Han surname. Some example of Hezhou Hui who became Salars are the Chenjia (Chen family) and Majia (Ma family) villages in Altiuli where the Chen and Ma families are Salars who admit their Hui ancestry. Marriage ceremonies, funerals, birth rites and prayer were shared by both Salar and Hui as they intermarried and shared the same religion since more and more Hui moved into the Salar areas on both banks of the Yellow river. Many Hui married Salars and eventually it became far more popular for Hui and Salar to intermarry due to both being Muslims than to non-Muslim Han, Mongols and Tibetans. The Salar language and culture however was highly impacted in the 14th-16th centuries in their original ethnogenesis by marriage with Mongol and Tibetan non-Muslims with many loanwords and grammatical influence by Mongol and Tibetan in their language. Salars were multilingual in Salar and Mongol and then in Chinese and Tibetan as they trade extensively in the Ming, Qing and Republic of China periods on the yellow river in Ningxia and Lanzhou in Gansu.[194]
Typhoid fever can cause a number of renal manifestations heretofore dubbed 'nephrotyphoid'. Haematuria in the absence of renal impairment is extremely rare among typhoid patients. We report a case of an adult who presented with a prolonged febrile illness and microscopic haematuria. Blood culture confirmed the diagnosis of typhoid and the patient was treated successfully with ceftriaxone. The Author(s) 2014 Reprints and permissions: sagepub.co.uk/journalsPermissions.nav.
fever maya banks epub 12
The Crimean-Congo hemorrhagic fever is a zoonotic disease transmitted by ticks and is characterized by fever and bleeding. It was seen for the first time in the south of present day Ukraine and thus named, Crimean fever. 1 In 1956, the virus was isolated in a patient with similar symptoms residing in Congo, Kenya and the virus was named Congo virus. The viruses causing these two diseases were the same and hence was termed Crimean-Congo hemorrhagic fever virus (CCHFV). Humans are the only known host that develops disease. Copyright Bentham Science Publishers; For any queries, please email at epub@benthamscience.org.
African swine fever (ASF) is an acute and often fatal disease affecting domestic pigs and wild boar, with severe economic consequences for affected countries. ASF is endemic in sub-Saharan Africa and the island of Sardinia, Italy. Since 2007, the virus emerged in the republic of Georgia, and since then spread throughout the Caucasus region and Russia. Outbreaks have also been reported in Belarus, Ukraine, Lithuania, Latvia, Estonia, Romania, Moldova, Czech Republic, and Poland, threatening neighboring West European countries. The causative agent, the African swine fever virus (ASFV), is a large, enveloped, double-stranded DNA virus that enters the cell by macropinocytosis and a clathrin-dependent mechanism. African Swine Fever Virus is able to interfere with various cellular signaling pathways resulting in immunomodulation, thus making the development of an efficacious vaccine very challenging. Inactivated preparations of African Swine Fever Virus do not confer protection, and the role of antibodies in protection remains unclear. The use of live-attenuated vaccines, although rendering suitable levels of protection, presents difficulties due to safety and side effects in the vaccinated animals. Several African Swine Fever Virus proteins have been reported to induce neutralizing antibodies in immunized pigs, and vaccination strategies based on DNA vaccines and recombinant proteins have also been explored, however, without being very successful. The complexity of the virus particle and the ability of the virus to modulate host immune responses are most likely the reason for this failure. Furthermore, no permanent cell lines able to sustain productive virus infection by both virulent and naturally attenuated African Swine Fever Virus strains exist so far, thus impairing basic research and the commercial production of attenuated vaccine candidates. 2018 Elsevier Inc. All rights reserved. 2ff7e9595c
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